Total Protein Assay Kit

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$120.00

Summary & Explanation

Plasma proteins are synthesized predominantly in the liver, plasma cells, lymph nodes, the spleen and in bone marrow. In the course of disease the total protein concentration and also the percentage represented by individual fractions can significantly deviate from normal values. Hypoproteinemia can be caused by diseases and disorders such as loss of blood, sprue, nephrotic syndrome, severe burns, salt retention syndrome and Kwashiorkor (acute protein deficiency). Hyperproteinemia can be observed in cases of severe dehydration and illnesses such as multiple myeloma. Changes in the relative percentage of plasma proteins can be due to a change in the percentage of one plasma protein fraction. Often in such cases the amount of total protein does not change. The A/G-ratio is commonly used as an index of the distribution of albumin and globulin fractions. Marked changes in this ratio can be observed in cirrhosis of the liver, glomerulonephritis, nephrotic syndrome, acute hepatitis, lupus erythematosus as well as in certain acute and chronic inflammations. Total protein measurements are used in the diagnosis and treatment of a variety of diseases involving the liver, kidney, or bone marrow, as well as other metabolic or nutritional disorders.

Category: Colorimetric Kits

Description

 

CATALOG # OttoBC154
PRODUCT NAME Urea Assay Kit
APPLICATIONS Fully automated analyzer, Spectrophotometer
REACTIVITY Universal
SAMPLES TYPE Serum, Plasma (Edta or Heparin)
Content Explanation Shelf life
Reagent-1 1x30ml 6 months
Calibrator 1×0.5ml 6 months
Q.Control 1×0.5ml 6 months

References

  1. Journal of the American College of Nutrition, Vol. 19, No. 6, 715–737 (2000) Published by the American College of Nutrition

 

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  1. Standing Committee on the Scientific Evaluation of Dietary Reference Intakes Food and Nutrition Board, Institute of Medicine: “Dietary Reference Intakes for Calcium, Phosphorus, Magnesium, Vitamin D and Fluoride.” Washington, DC: National Academy Press, 1997.

 

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